Menghon Cheah

Menghon Cheah

Visiting Research Associate


Menghon joined the CDN (Grubb lab) in April 2017. His BBSRC-funded project is to investigate the connectivity of adult-born dopaminergic neurons in the olfactory bulb. Before joining the lab, he was at the University of Cambridge doing a PhD in Clinical Neurosciences focusing on axon regeneration and spinal cord injury under the supervision of Prof. James Fawcett and funded by the Cambridge Trusts. Since 2016, he has been an elected member of the Royal Society of Biology (MRSB). He was thrilled to be back at his alma mater, King’s, where he studied BSc (Hons) in Neuroscience and graduated with First Class Honours. During the second year of his undergraduate, he spent the entire year as an exchange student at the University of California in San Diego.


1. Cheah M, Fawcett JW, Andrews MR (2017) Assessment of Thermal Pain Sensation in Rats and Mice Using the Hargreaves Test. Bio-protocol 7(16): e2506.

2. Cheah M, Fawcett JW, Andrews MR (2017) Dorsal root ganglion injection and dorsal root crush injury as a model for sensory axon regeneration. J. Vis. Exp. (123), e55535.

3. Cheah M, Fawcett JW, Haenzi B (2017) Differential regenerative ability of sensory and motor neurons. Neurosci Lett. 652:35-40.

4. Cheah M, Andrews MR (2016) Targeting cell surface receptors for axon regeneration in the central nervous system. Neural Regen Res 11(12):1884-1887.

5. Cheah M, Andrews MR, Chew DJ, Moloney E, Verhaagen J, Fässler R, Fawcett JW (2016) Expression of an activated integrin promotes long-distance sensory axon regeneration in the spinal cord. J Neurosci 36(27):7283-97.

6. Andrews MR, Solemon S, Cheah M, Mason M, Moloney E, Verhaagen J, Bensadoun J, Schneider B, Aebischer P, Fawcett JW (2016) Axonal localization of integrins in the CNS is neuronal type and age dependent. eNeuro 3(4) e0029-16.2016.

7. Kappagantula S, Andrews MR, Cheah M, Abad-Rodriguez J, Dotti CG, Fawcett JW (2014) Neu3 sialidase-mediated ganglioside conversion is necessary for axon regeneration and is blocked in CNS axons. J Neurosci 34(7):2477-92.


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